tag:blogger.com,1999:blog-36840063.post3848507466084228723..comments2024-03-27T22:57:00.742+00:00Comments on Hyperlipid: The thumb tack hypothesisPeterhttp://www.blogger.com/profile/14527788116058656094noreply@blogger.comBlogger46125tag:blogger.com,1999:blog-36840063.post-13412771791720511982009-10-26T21:48:33.859+00:002009-10-26T21:48:33.859+00:00thanks everyone for the input but only Fred has ac...thanks everyone for the input but only Fred has actually addressed the protein issue. I understand the value of high fat, and already eat this way. My query is the scientific understanding as to why too much protein is damaging (personally vested since I eat a lot of meat)? I always try to choose fatty cuts but I still eat over 100g of protein a day.<br />LeenaS- Thanks, that makes sense.mathmanhttps://www.blogger.com/profile/12093059295621099894noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-36027986628767850322009-10-25T20:16:50.604+00:002009-10-25T20:16:50.604+00:00Gunther (and Bamboo),
Thanks for the comment, pai...Gunther (and Bamboo),<br /><br />Thanks for the comment, pain free I hope!<br /><br />With Bamboo's comment on the <a href="http://high-fat-nutrition.blogspot.com/2008/03/kwasniewski-praise-lard.html" rel="nofollow" rel="nofollow">Praise the Lard</a> post I think it is timely to run through the biochemistry we discussed as a post. Might get some time tomorrow but after that it's 3 12 hour dayshifts coming up so...<br /><br />PeterPeterhttps://www.blogger.com/profile/14527788116058656094noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-4789166561377699212009-10-25T19:56:43.292+00:002009-10-25T19:56:43.292+00:00Fred Hahn
JK is Jan Kwasniewski and his diet is t...Fred Hahn<br /><br />JK is Jan Kwasniewski and his diet is the Optimal Diet. Peter talks about it in the "Food" and "What I Eat" blog categories. Basically it is an 80% animal fat diet. If you follow Peter's additional recommendations, you also take out grains, add omega 3 and tweak a few other things.<br /><br />Mathman,<br /><br />I've been on OD for 2 years. Besides losing the weight I wanted to lose almost effortlessly (6 months with no hunger), I have also seen a resolution of many symptoms of immune system breakdown, such as stoppage of hair loss, disappearance of chills and low body temp, resolution of Reynauds, and off-blog, Peter and I have discussed whether a recent kidney stone incident I had was actually the OD SHRINKING the kidney stones in my kidneys over the last 2 years so that they could finally pass down the ureter. In other words, the OD didn't cause the stones, it finally got the small enough to get rid of.<br /><br />Of course, we'll never know if that's the case, but I really haven't slept as well or had as much energy before I started the plan.<br /><br />Full disclosure: Not selling anything and have never even met Jan Kwasniewski or Peter personally! <br /><br />Hope that helps.gunther gathererhttps://www.blogger.com/profile/15361732213105267048noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-36944329571368272472009-10-25T11:48:12.810+00:002009-10-25T11:48:12.810+00:00Mathman said:
"I would love to hear Peter...Mathman said:<br /><br />"I would love to hear Peter's thoughts or anyone else following a Jan K.-type of diet. I have been hearing more and more that the carbs and fats are interchangable for optimal health so long as protein remains low-why?"<br /><br />I don't know what Jan K. is but research pretty much shows that if protein intake is optimal, you can swap fat for carbs for weight loss, not for optimal health. <br /><br />Yes we can indeed get fatter eating only fat and protein in excess of our caloric needs. However, I doubt one could become obese eating like this.Fred Hahnhttps://www.blogger.com/profile/01287565594918201935noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-47912854535090916272009-10-25T03:41:24.882+00:002009-10-25T03:41:24.882+00:00medium-chain...
Fat does put on fat in certain am...medium-chain...<br /><br />Fat does put on fat in certain amounts, thank god. Fat needs to put on fat, or else we'd have died out a long time ago. We seem to all accept that our ancestors had to tap into those stores of bottom-fat on a regular basis, how is this possible (or anything other than deadly) unless we are quickly able to restore those stores of bottom-fat?? In-fact, putting on fat from fat is the only conclusion to draw if you think eating fat is an adaption in humans.<br /><br />other person:<br />carbs or fat calories, which is going to give you a powerhouse of fat soluble vitamins? what does bread give you?Jocelynhttps://www.blogger.com/profile/08944759372390475805noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-49429170496141972052009-10-24T14:59:19.321+00:002009-10-24T14:59:19.321+00:00Mathman:
As for your latter question, the mitocon...Mathman: <br />As for your latter question, the mitocondria count. According to Nick Lane more is better; much better.<br /><br />More means smaller work load for each mitochondria. Which means less leaking electrons and better control on the general quality of the thousands of mitochondia units in each and every cell.<br /><br />And as for Dr. Lane, he seems to be getting quite a bit of respect on his wievs, and not just articles in Science:<br />http://blogs.sciencemag.org/origins/2009/10/prizewinning-researcher-ventur.html<br />Very interesting work indeed (thanks peter for this!)<br /><br />LeenaSLeenaShttps://www.blogger.com/profile/09565398001624904475noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-53589416701081893152009-10-24T00:27:13.909+00:002009-10-24T00:27:13.909+00:00I would love to hear Peter's thoughts or anyon...I would love to hear Peter's thoughts or anyone else following a Jan K.-type of diet. I have been hearing more and more that the carbs and fats are interchangable for optimal health so long as protein remains low-why?<br /><br />Also, from a longevity standpoint is more mitochondria better? This seems like the "stoking the fire" analogy which speeds cell processes and creates more oxidative damage.mathmanhttps://www.blogger.com/profile/12093059295621099894noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-35964408224101327372009-10-23T19:21:58.914+00:002009-10-23T19:21:58.914+00:00hate to do this, but since I realized the nutritio...hate to do this, but since I realized the nutrition links aren't working, I put the images of the nutrition facts <a href="http://www.medium-chain-triglycerides.com/2009/10/nutrition-facts.html" rel="nofollow">here</a>.Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-36840063.post-49310625894725812922009-10-23T18:57:07.229+00:002009-10-23T18:57:07.229+00:00whether or not this sounds doubtful, the fact of t...whether or not this sounds doubtful, the fact of the matter is, for a period of about 3-4 weeks (until I was certain this diet was putting on weight), I consumed 4-6 sticks of butter / 1-1.5 lbs of beef tallow (tissue and oil); meat was held at 1-1.5 lbs beef chuck.<br /><br />perhaps 90% was too high an estimate, so let's break down the numbers:<br /><br />typical day:<br /><a href="http://fatsecret.com/Diary.aspx?pa=fjrd&eid=11007908&dt=14533" rel="nofollow">beef chuck 16oz</a>, 86g protein, 429 fat kcal, 798 kcal<br /><a href="http://fatsecret.com/Diary.aspx?pa=fjrd&eid=11006536&dt=14533" rel="nofollow">4 sticks butter</a> 0.27g carbs, 367g fat, 3241 fat kcal<br /><br />(429+3241)/(798+3241) = 0.9086<br /><br />If it was beef tallow, I consumed both the tissue and the oil, so let's estimate 1 lb fat = 3500 kcal,that's about 3500-5250.<br /><br />I'm 150lb, 5-9, low-active. I probably didn't need all those calories.<br /><br />That's what happens when I'm told that fat can't make fat.<br /><br />I see this was asked <a href="http://www.paleonu.com/panu-weblog/2009/8/25/on-zero-carbs-can-you-make-your-glucose-from-fat.html" rel="nofollow">here</a> in the comments.Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-36840063.post-70645595781775625822009-10-23T10:52:19.895+00:002009-10-23T10:52:19.895+00:00"What is the main problem with protein? I wan..."What is the main problem with protein? I wanted to know why eating close to a gram per pound of LBM would be bad. Is it the insulin?"<br /><br />It isn't bad and if you are weight lifting it should be more in the realm of 1.2gms per pound of lean body mass. <br /><br />Eating this much protein is not bad so long as your carbs are low. Glucagon 'offsets' so to speak or rather takes care of the rise in insulin from higher protein intakes. This is best described in books by Drs. Eades and by Taubes.Fred Hahnhttps://www.blogger.com/profile/01287565594918201935noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-69913921527384131762009-10-23T03:16:23.058+00:002009-10-23T03:16:23.058+00:00Peter -
What is the main problem with protein? ...Peter - <br /><br />What is the main problem with protein? I wanted to know why eating close to a gram per pound of LBM would be bad. Is it the insulin?mathmanhttps://www.blogger.com/profile/12093059295621099894noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-73379719472810448582009-10-22T16:16:52.856+00:002009-10-22T16:16:52.856+00:00Moving Too Little or Eating Too Much? .
How much p...<a href="http://www.psychologytoday.com/blog/the-human-beast/200909/moving-too-little-or-eating-too-much" rel="nofollow">Moving Too Little or Eating Too Much? </a>.<br /><a href="http://www.psychologytoday.com/blog/the-human-beast/200910/how-much-physical-activity-do-we-really-need" rel="nofollow">How much physical activity do we really need? </a>Kenhttps://www.blogger.com/profile/01637818790791725275noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-10161706932527060112009-10-22T13:00:08.462+00:002009-10-22T13:00:08.462+00:00OK, back on blog for a while!
No carb and Fred, I...OK, back on blog for a while!<br /><br />No carb and Fred, I think there are subtle differences between the type one diabetic and the no carber/all fatter. These are thoughts, not evidence based.<br /><br />The first is that we always have some glucose reserve, even if it is only in protein. If we manage to eat hypercaloric amounts of fat but cannot store it there will be an oversupply of fatty acids. If the liver has enough fatty acids it will become physiologically insulin resistant and secrete a little glucose. This glucose will provoke an insulin rise. It only takes a little insulin to store fat. When nothing is broken this is how I see it should work. I can't see that total waste of excess fat calories is what would happen.<br /><br />In the type one diabetic there would be no compensatory secretion of insulin and glucose would rise while free fatty acids continued to cause glucose rejection. Without the supplementary insulin, glucose and fatty acids would rise until we get the classic diabetic ketoacidosis.<br /><br />What probably determines outcome is the residual ability of a type 1 diabetic pancreas to muster enough insulin to respond to the physiological signaling through hepatic glucose secretion in response to FFAs making it ignore insulin's signal to hold on to glycogen. However carb restricted type 1 diabetics were, pre insulin, they did usually die in the medium term unless they had some residual pancreatic function left.<br /><br />Bris, I'd agree with Fred, Cordain has said some unbelievably stupid things on occasions. This smells like one of them!<br /><br />JB, I think we store all fat as adipose tissue all the time. Even from meal to meal fat moves from the gut to adipose tissue, even if it's only for a few hours. Otherwise LC/high fat eaters would be massively hypertriglyceridaemic from chylomicrons. It gets packed away. The beauty of LC is that it gets unpacked just as easily, without hunger...<br /><br />Matt, I think the above is how I see the physiology, but again it's not hard data. People rarely measure what you want them to!<br /><br />Mikael,<br /><br />As I see it the number is relatively unimportant compared to the activity. When I started following this after your initial query what came out was that mitochondria are not organelles, they are more like living entities in their own right and they are a heterogenous bunch. Some are powerhouses and some are duds and there is s sort of sex to share DNA to minimse the number of duds. That might be why there is so little emphasis on numbers in most studies. Dr BG's links and Fred's comments are both very perceptive in this respect. <br /><br />To query the assertion that high fat diets increase mitochondrial numbers in humans is excellent as a regulator of unsubstantiated statements. Let's say they do in mice. But I personally would be amazed if mice mitochondria responded differently to human mitochondria. I've even got time for C. elegans mitochondria being pretty much the same as human ones...<br /><br />Peter<br /><br />With appologies, this is probably (even more) full of typos (than usual)!Peterhttps://www.blogger.com/profile/14527788116058656094noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-84340046340106431242009-10-21T11:06:14.023+00:002009-10-21T11:06:14.023+00:00In this study:
"The effect of strength trai...In this study: <br /><br />"The effect of strength training on estimates of mitochondrial density and distribution throughout muscle Fibres" <br /><br />Philip D. Chilibeck á Daniel G. Syrotuik á Gordon J. Bell<br />Eur J Appl Physiol (1999) 80: 604±609<br /><br />If you calculate the numbers properly you'll see that though mitochondrial density decreased as a result of myofibular hypertrophy the total volume of mitochondria increased significanlty. <br /><br />Also, endurance training can lead to skeletal muscle loss which can be seen under the micrscope as an increase in mitochondrial density but which is not positive.Fred Hahnhttps://www.blogger.com/profile/01287565594918201935noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-74346072110766311072009-10-21T08:40:27.445+00:002009-10-21T08:40:27.445+00:00Well, thanks, but I'm looking for evidence on ...Well, thanks, but I'm looking for evidence on high-fat diets increasing the number/density of mitochondria <em>without</em> added exercise.<br /><br />That's what's being claimed, so that's what needs to be proven. Or is the claim pulled out of thin air?Unknownhttps://www.blogger.com/profile/14446728887985553139noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-38492874825886128982009-10-20T21:23:09.104+00:002009-10-20T21:23:09.104+00:00It's been known since the 1970s... but recentl...It's been known since the 1970s... but recently... you have to use 'em or your lose 'em! There are physiological reasons to 'taper' prior to a half-marathon or other events. The intramyofibrillar mitochondria only increase in quantity preferentially toward the END of training, non-linearly. Dr. Fluck has interesting thoughts (*haa). A high fat diet can prime the mitochondria... but not raise the mitochondria. Got use it.<br /><br />The effect of concurrent endurance and strength training on quantitative estimates of subsarcolemmal and intermyofibrillar mitochondria.<br />Chilibeck PD, Syrotuik DG, Bell GJ.<br /><br />Int J Sports Med. 2002 Jan;23(1):33-9.<br /><br /><br />Diabetics:<br />http://www.newswise.com/articles/even-at-advanced-age-exercise-boosts-muscle-cells-energy-centers<br /><br /><br /><br />Med Sci Sports Exerc. 2003 Jan;35(1):95-104. Links<br />Plasticity of skeletal muscle mitochondria: structure and function.Hoppeler H, Fluck M.<br />Department of Anatomy, University of Bern, Switzerland. hoppeler@ana.unibe.ch<br /><br />Mitochondria in skeletal muscle tissue can undergo rapid and characteristic changes as a consequence of manipulations of muscle use and environmental conditions. Endurance exercise training leads to increases of mitochondrial volume of up to 50% in training interventions of a few weeks in previously untrained subjects. Additionally, a shift of substrate metabolism toward a higher reliance on lipids is observed, structurally reflected as a doubling of the intramyocellular lipid content. A similar increase in intramyocellular lipids without an increase in mitochondrial volume is observed as a consequence of a high-fat diet. Strength training has a major impact on muscle myofibrillar volume, however the mitochondrial compartment appears relatively unchanged. Bedrest and microgravity conditions lead to losses of both myofibrillar and mitochondrial volume, likely as a consequence of the decrease in metabolic and mechanical stress on muscle tissue. Permanent severe hypoxia leads to a loss of muscle mass and muscle oxidative capacity; however, hypoxia signaling events are triggered, which lead to distinct reprogramming phenomena of the transcriptome of the muscle cells. The molecular mechanisms that orchestrate the plasticity of skeletal muscle mitochondria are just beginning to unfold. The present data indicate that transcriptional events largely contribute to increases in mitochondrial mass in human skeletal muscle with endurance training. Expression of mitochondrial proteins from the nuclear and mitochondrial genomes is coordinated and involves the nuclear-encoded transcription factors NRF-1 and TFAM. Transcription of genes encoding the mitochondrial proteins involved in beta oxidation can be regulated separately from the genes of the Krebs cycle and the respiratory chain. Transcription factors AP-1 and PPARalpha/gamma and the protein kinase AMPK are signaling molecules that transduce the metabolic and mechanical factors sensed during endurance training into the complex transcriptional adaptations of mitochondrial proteins.<br /><br />PMID: 12544642Dr. B Ghttps://www.blogger.com/profile/15451872961651116061noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-4323297342519486142009-10-20T19:41:48.571+00:002009-10-20T19:41:48.571+00:00J Am Coll Cardiol. 1997 Apr;29(5):1067-73.
Effec...J Am Coll Cardiol. 1997 Apr;29(5):1067-73. <br /><br />Effects of endurance training on mitochondrial ultrastructure and fiber type distribution in skeletal muscle of patients with stable chronic heart failure. Hambrecht R, et al.<br /><br />OBJECTIVES: The present study was designed to evaluate the effects of an ambulatory training program in patients with chronic heart failure (CHF) on the ultrastructural morphology of mitochondria and fiber type distribution of skeletal muscle and its relation to peripheral perfusion. <br />BACKGROUND: Recent studies in patients with CHF have suggested that intrinsic abnormalities in skeletal muscle can contribute to the development of early lactic acidosis and fatigue during exercise. <br />METHODS; Patients were prospectively randomized to either a training group (n = 9; mean [+/- SD] left ventricular ejection fraction [LVEF] 26 +/- 10) participating in an ambulatory training program or to a physically inactive control group (n = 9; LVEF 28 +/- 10%). At baseline and after 6 months, patients underwent symptom-limited bicycle exercise testing with measurement of central and peripheral hemodynamic variables as well as percutaneous needle biopsies of the vastus lateralis muscle. The mitochondrial ultrastructure of skeletal muscle was analyzed by ultrastructural morphometry; cytochrome c oxidase activity was visualized by histochemistry and subsequently quantitated by morphometry. The fiber type distribution was determined by adenosine triphosphatase staining. <br />RESULTS: After 6 months of exercise training there was a significant increase of 41% in the surface density of cytochrome c oxidase-positive mitochondria (SVMOcox+) (p < 0.05 vs. control) and of 43% in the surface density of mitochondrial cristae (SVMC) (p < 0.05 vs. control). Furthermore, exercise training induced a 92% increase in the surface density of the mitochondrial inner border membrane (p < 0.05 vs. control). In contrast, the total number of cytochrome c oxidase-positive mitochondria remained essentially unchanged. Exercise-induced improvement in peak oxygen uptake was closely linked to changes in SVMOcox+ (p < 0.01, r = 0.66). After exercise training, changes in submaximal femoral venous lactate levels were not related to changes in submaximal leg blood flow (r = -0.4), but were inversely related to changes in the volume density of mitochondria (p = 0.01; r = -0.6) as well as to changes in SVMC (p < 0.05; r = -0.5). After exercise training there was a "reshift" from type II to type I fibers (p < 0.05 vs. control). CONCLUSIONS: Patients with CHF who engage in regular physical exercise show enhanced oxidative enzyme activity in the working skeletal muscle and a concomitant reshift to type I fibers. These exercise-induced changes in oxidative capacity appear to be unrelated to changes in peripheral perfusion.<br /><br />PMID: 9120161Dr. B Ghttps://www.blogger.com/profile/15451872961651116061noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-17211732234580435062009-10-20T07:58:27.525+00:002009-10-20T07:58:27.525+00:00and the mitochondrial quantity change accordingly ...<em>and the mitochondrial quantity change accordingly as well.</em><br /><br />Do you have studies on humans as opposed to rats showing this?Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-36840063.post-50151406444648161172009-10-19T19:29:32.846+00:002009-10-19T19:29:32.846+00:00The insulin component is so true though, and it ta...The insulin component is so true though, and it takes such a tiny fraction of calories to be diverted into fat storage compared to that burned to create a steady increase in total body fat. <br /><br />For example, the energy found in a single peanut being stored in body fat on a daily basis is enough to make a person gain the typical 40 pounds between ages 20 and 60. It's not something we could ever consciously regulate.Matt Stonehttps://www.blogger.com/profile/00823163098708883587noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-83468471387682160492009-10-19T19:26:36.736+00:002009-10-19T19:26:36.736+00:00Good post. I can't believe that those fatties...Good post. I can't believe that those fatties are moving that much. I'd be lucky if I moved 37 minutes a day. 370? <br /><br />There are a couple of fallacies in the post though, one is that BMR can't change much. The range of BMR is -50% on the downside, meaning you can go from burning 3,000 at stable weight to burning 1,500 on stable weight. <br /><br />On the topside, you can increase BMR by 100%, meaning you can maintain weight on 6,000 vs. a normal 3,000. <br /><br />It's a biological fundamental that very few are hip to, but that studies such as Ancel Keys' starvation study and Ethan Sims overfeeding has shown very conclusively.<br /><br />Overfeeding also does not raise insulin levels, but actually decreases insulin resistance and leptin resistance and lowers insulin levels, which is why overfeeding leads to massive decreases in hunger and rapid weight loss when overfeeding stops. It's a mirror to the effects of underfeeding. <br /><br />Overfeeding can actually be quite metabolically healing, and builds tremendous digestive strength.Matt Stonehttps://www.blogger.com/profile/00823163098708883587noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-1194440759064003232009-10-19T18:28:03.555+00:002009-10-19T18:28:03.555+00:00Actually the human body's efficiency adapts to...Actually the human body's efficiency adapts to demand -- more demand (high intensity short duration v. low intensity long duration) changes the genetic expression of different receptors in the muscles and hormones. Different muscles fibers are favored dep on the demand (fast twitch v. slow twitch) and the mitochondrial quantity change accordingly as well.<br /><br />This has been shown in studies... as well as the many who follow Paleo type ancestral exercises! Personally noticed I eat less and have more energy -- could it be I have better hormones and better insulin and better efficiency? Yes -- it is all modifiable. Hormones and demand...Dr. B Ghttps://www.blogger.com/profile/15451872961651116061noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-69316327462491814562009-10-19T14:44:09.851+00:002009-10-19T14:44:09.851+00:00Fred/All,
Some time ago, I think it was Arthur De...Fred/All,<br /><br />Some time ago, I think it was Arthur DeVany who posited the idea from his research that a paleolithic man might have had a "2X" or "3X" lifestyle meaning that he/she burned, through the rigors of a tough, demanding life, two or three times base calories needs. So, if 1,500 calories was the bogey, then 3,000 to 4,500 owing to their heightened activities.<br /><br />My guess is if this is accurate, it ensures that using stored bodyfat as the source for a lot of those calories on many days was essential between successful hunts.<br /><br />Yes, to lots of calories ending up as heat...we are warm blooded and regulate such matters. Where heat signals wasted energy in some systems, maybe less so for systems depending on it?<br /><br />For me, the idea that a man weighing 175 lbs. can ride a bike from Dallas, to Houston, back to Dallas, and back to Houston... on 8 or 9 lbs. of bodyfat... it is still pretty impressive.<br /><br />Other than a personal general belief that about 2/3s or more of one's food should be fat calories of the right sorts, then carbs and protein, it seems to me that the real goal is to optimize, via insulin and other factors, the ability to get excess sugar out of the blood where it is harmful, then to be able to get energy out of stored fat, when blood sugar is too low.<br /><br />BradBrad Reidhttps://www.blogger.com/profile/10275394962283072954noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-63049479084309824322009-10-19T11:14:44.924+00:002009-10-19T11:14:44.924+00:00"Wrong. Before insulin was discovered in the ..."Wrong. Before insulin was discovered in the 1920s Type 1 diabetics (who don't produce any insulin) were placed on zero-carbohydrate meat-based diets. They gained weight on these diets."<br /><br />Reference please. I think you are missing the point howevere Bris. I am sure that these diabetics you mention were given insulin. But the fact remains, no insulinm no fat storage. That is not debatable. <br /><br />All the body knows is fat, carbs and protein. You will NOT necessarily get loose stools eating an 80% fat diet. If so it will be temporary. But 80% is an arbitrary number you made up. Most paelo-eating folks stick to a 60-70% fat diet, 20-30% protein 10-20%carb eating plan. <br /><br />"Humans instinctively match daily calorie intake with energy expenditure to within ~0.2% regardless of diet. This is why it usually takes decades to become severely overweight."<br /><br />Reference please. Is the loss of lean tissue as the years roll on figured into this 'equation?'<br /><br />"Low carb diets cause weight loss via reduced calorie intake due to reduced appetite." <br /><br />Since it is true that some people can indeed take in more calories than their BMR and not gain fat and since some can do the opposite, your statement is false. I probably don't need to reference the research in the book 'Good Calories Bad Calories' for support, right? <br /><br />And remember, weight loss and fat loss are very different. <br /><br />"The body cannot tolerate high amounts of protein (over 5g/kg bodyweight)." <br /><br />Even if this is true (which I don't believe it is - reference please) so what? Who's going to eat that much protein? Even hardcore bodybuilders don't eat this much.<br /><br />Bris - obesity is a disorder of excess fat accumulation casued by a disruption in the hormonal mechanisms of the human body caused by excessive insulin production (among other things). <br /><br />You don't look at a 6'6", 10 year old boy and say - "Hey look - he's really eating too much and he's lazy!" Hormones are out of control. You don't watch a pregnant woman who is bawling over her choice of ice cream at Ben and Jerry's and say "She's a nutjob." Hormones are raging. <br /><br />Hormones, not total calories are the deeper picture. We need to be looking more deeply into the obesity issue especially for children.Fred Hahnhttps://www.blogger.com/profile/01287565594918201935noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-14776424792690864422009-10-19T10:55:52.139+00:002009-10-19T10:55:52.139+00:00"Loren Cordain has stated that "adequate..."Loren Cordain has stated that "adequate" exercise for a male is 'running 15 miles wearing a 25lb backpack every day."<br /><br />This amount of exercise is absurd. Humans are not designed - no animal for that matter is designed - for an amount of exercise and punishment. <br /><br />Maintaining a normal body weight is all about eating real food. Exercise should be for maintaining and/or increasing lean tissue.Fred Hahnhttps://www.blogger.com/profile/01287565594918201935noreply@blogger.comtag:blogger.com,1999:blog-36840063.post-26588942158698346932009-10-19T10:38:53.723+00:002009-10-19T10:38:53.723+00:00JB said:
"Peter, it is just really tough to ...JB said:<br /><br />"Peter, it is just really tough to lose weight via exercise because of our bodies' extreme operating efficiency."<br /><br />There is nothing efficient about the human body's energy use. A world class road cyclist can only produce 0.5 hp for one hour. 80% of our energy output is in the form of heat. <br /><br />The only reason why exercise is "ineffective" is that very few people ever do anywhere near enough to lose weight. Jogging for 30 minutes a day is only going to burn 1kg of fat in a month. <br /><br />Elite athletes are often forced to gorge themselves constantly to maintain their weight. <br /><br />Our HG ancestors would have been burning at least 1000Cal a day in physical activity over their BMR. This is equivalent to burning 50kg of fat per year. <br /><br />Loren Cordain has stated that "adequate" exercise for a male is 'running 15 miles wearing a 25lb backpack every day.'Brishttps://www.blogger.com/profile/05771534538377496202noreply@blogger.com