Tuesday, July 08, 2008

Statin stupidity again

Read and cry.

Cure for C sections!

Perhaps a little less sugar might be a better approach!

I found this while searching for a snippet from Radio 4's Today Program announcing the recommendation from field leaders in the USA that children should start taking stains at 8 years of age, on the off chance it might eliminate all illness for ever. Happy happy happy I don't think. Couldn't find it on R4, but it'll surface soon in other places I guess.



Edit: You've all read Chris's post here? Never mind which end of life, you have a statin deficiency.


Unknown said...

Look no longer for your snippet... here's a NYT article (plus a NYT blog response) on statins for kids:


Peter said...

Hi Jenny,

Thanks for ending the search, not sure if it makes me any happier!!!!!!



Rita. said...

The AAP is now recommending statins for children. I never thought I'd see the day.

Anonymous said...

I think you have to go back in time to eliminate the risk of C-section. Women can never give birth normally if the pelvis didn't grow properly, because of poor nutrition. If drugs have any benefit, they are probably from causing premature birth or low birth weight. The most telling line in the article was this:
"Statins are already widely prescribed to cut the risk of heart disease.
But they are not recommended for women who are pregnant or breastfeeding, as there is concern of a possible link to birth defects."

They are trying to get everybody on statins, while ignoring what really causes heart diseases - junk carbs, junk fats, stress, inactivity, lack of sleep, lack of sunlight, etc. An elevated cholesterol level is just a marker of the damage being caused to the arteries and blood vessel by eating the typical modern diet and being a sedentary shut-in.

Momzuki said...

It is well known among farm-folk that animals that get too fat have a hard time giving birth. You can feel the extra fat in the birth canal of say, a horse. Of course the extra fat cushions the contractions.

frank said...

The full press release is here: http://tinyurl.com/6fu69x

The American Academy of Pediatrics has issued new cholesterol screening and treatment recommendations for children. The policy statement, “Lipid Screening and Cardiovascular Health in Childhood,” recommends cholesterol screening of children and adolescents with a family history of high cholesterol or heart disease. It also recommends screening patients whose family history is unknown or those who have other factors for heart disease including obesity, high blood pressure or diabetes. Screening should take place after age two, but no later than age 10. The best method for testing is a fasting lipid profile. If a child has values within the normal range, testing should be repeated in three to five years. For children who are more than eight years old and who have high LDL concentrations, cholesterol-reducing medications should be considered. Younger patients with elevated cholesterol readings should focus on weight reduction and increased activity while receiving nutritional counseling. The statement also recommends the use of reduced-fat dairy products, such as two percent milk, for children as young as one year of age for whom overweight or obesity is a concern.”

For children over 8, notice there is no recommendation for lifestyle measures!

There are many issues of concern with consensus-based guidelines especially regarding the “treatment” of children with drugs with proven toxicity. As adults we can do our own research and refuse such madness, or decide the side effects are intolerable and throw them in the bin, but lawmakers and child protection services use these guidelines to enforce medication (it already happens in cancer, ADHD, ASD, HIV and obesity) or they forcibly remove the children. These guidelines are the “final word” uttered by experts – no science required, and are used in court to prove “parental abuse” in not giving said medication. This isn’t theoretical – it’s happening already.
Children do not develop fatty streaks of any significance until age 19 – how does lowering cholesterol in 1999 prevent plaque formation in 2008?
It is another tragedy in the making and speaks to the desperation of an industry manipulating puppet science to create new markets in the name of profits. The next move will be to arbitrarily lower the “normal” range for cholesterol in children as they have done several times in adults. Insert expletives here________.

frank said...

My 1991 ABPI Data Sheet Compendium gives normal range total cholesterol as 2.8 – 7.8 mmol/L, and as you know the ULN is now only 5 mmol/L with no lower cut-off point. Looking at the 1998 levels of 80,000 people in a large metropolitan city almost nobody falls into the hypercholesterolaemia label under the old values, but with the new values 70% of the 45-70 year bracket do!
The National Cholesterol Education Program's Expert Panel on Blood Cholesterol in Children and Adolescents give total cholesterol reference values in children (2-19 years) as < 170 mg/dl, which converts to 4.6 mmol/L. I couldn’t find the full report available, which annoys the tits off me as it was funded from the public purse, but found a non scientifically referenced précis of the report from American Family Physician stating the 75th percentile for TC in children is (the magic number!) 170 mg/dl. Put another way that’s 25% of ALL children who will be deemed in need of “treatment”.
From an AAP 1998 statement: http://tinyurl.com/6kwa6o
“Because no long-term studies of the relationship of blood cholesterol levels measured in childhood to coronary heart disease in later life have been conducted, the relationship of childhood cholesterol levels to the atherosclerotic process must be inferred from less direct evidence.”
So “we don’t know what cholesterol level range is ‘normal’ for children, and we don’t know at what level it should be deemed “pathological” so we will arbitrarily define 25% of all children as being hypercholesterolaemic and in need of treatment with drugs (of questionable benefit and much evidence of harm) that have never been tested in healthy children based on a leap of faith that childhood cholesterol levels may in some unspecified way lead to CVD in later life.”
My disgust and contempt for these pharma puppets is registering a 10 on the Richter scale – these people better keep away from me or I might have to do something grievous with Vaseline and an angle-poise lamp!

Debs said...

Hmm, put them on medication early, especially medication which hasn't been tested on children, and for which long-term effects have been problematic in adults. Fabulous idea. Get rid of traditional foods too, and feed them lots of low-fat dairy.


The good news is, apparently there's been a lot of backlash from doctors (and everyone else)

Food Is Love

Anonymous said...

"It is well known among farm-folk that animals that get too fat have a hard time giving birth."

This is true, but we must also take into account malnutrition. Women of today often have a poorly developed pelvis, due to lack of naturla fats and proteins while growing, and an abundance of grains, refined carbs, and PUFA vegetable oils. Such women are not going to give birth easily, no matter what they eat. Like if a person's jaw doesn't grow properly, they're going to have crowding and impaction and decay. Primitives did not have this problem, because they ate natural foods and spend most of their day standing. Folks today eat processed garbage and spend most of their day sitting down.

TedHutchinson said...

Statins and Vitamin D
comments on this paper

Increased Levels of 25 Hydroxyvitamin D and 1,25-Dihydroxyvitamin D After Rosuvastatin Treatment: A Novel Pleiotropic Effect of Statins?Full text online.
This is likely to be the way the claimed clinical benefit of statins is achieved and may be the pleiotropic effect of rosuvastatin, decreasing mortality in patients with coronary artery disease.

But they still don't know the mechanism involved

This is a new listing on PUBMED and it's relevant here because we know of the Association between vitamin D deficiency and primary Cesarean section

It seems to me pointless taking a statin (with many obvious and dangerous side effects) that achieves it's magic by actually increasing your vitamin D3 status (by some mechanism as yet unidentified) when you can more cheaply and more effectively and without any side effects achieve the same effect (by taking Cholecalciferol Vitamin D3.

It is sheer madness (or the perverted logic of big Pharma) to make people take a drug that has potential for unpleasant side effects that works by mimicking a safe supplement that is cheap and doesn't produce side effects at the normal amounts most people reading this require.

Betsy said...

Peter, do you know why statins cause antibodies, I know two people who developed polymyositis simply from taking statins, and actually have antibodies as if it is an autoimmune condition, but without a pathogen.

And do you know any way to reverse what the statins have done? Both people are taking CoQ10 but one of them is also taking prednisone and methotrexate!!

So sad!

The worst part is my Mom is taking simvastatin and is degenerating so badly. I will get some CoQ10 to her, but it is almost useless to try to correct the problem while she is still taking it.

Unknown said...

Betsy, it seems one of the main reasons why statins cause antibodies is because they increase endotoxin exposure in the blood stream by way of lowering blood cholesterol content, increasing systemic inflammation mediated by various harmful agents, and because statins are little more than fungal toxins all by themselves (this is rather well explained in the scholarly well referenced article "Do Garcinia Cambogia Side Effects Boost Diabetes?" - direct link: at http://www.supplements-and-health.com/garcinia-cambogia-side-effects.html - look especially at Figure 4 & 8).

Peter said...

Betsy, all major tissue injury induces auto antibodies. This appear to be a normal response to injury. People with major trauma will occasionally go on to develop lupus or similar auto immune diseases. This appears to be an abnormal exaggeration of the normal process... http://www.ncbi.nlm.nih.gov/pubmed/7165361


Betsy said...

Thank you, Peter and Paul. (PS: Where's Mary? Or is that just an American joke?)

Anyway, thank you for the article, Paul. Very interesting on many levels. Very long, too. Peter, don't read this part.(What do you think about his thoughts on glucose burning cleaner than fatty acids?)

Peter, do the statins actually cause injury to tissue? The people with polymyositis said that the antibody is to HMGR. Do the antibodies attack the site where HMGR binds? I did read that once the antibody action is started, that's what keeps the whole process going after the statins are discontinued.

I didn't understand how statins actually affect HMG CoA, so I looked it up and found this site. http://proteopedia.org/wiki/index.php/User:Eran_Hodis/Sandbox7

The statins take the place of part of the HMGR protein, making HMGR no longer effective. I admit I have to read it again, and I know probably everyone else already knows this.

I also read that higher blood sugar levels activate more HMG CoA activity, so I'm wondering if slowing down that activity by controlling blood sugar might be one way to slow down the autoimmunity???

Betsy said...

Paul, with respect to statins being made with fungus, do you think they carry any of the endotoxins along with them? And if they do, would that contaminate the HMGR, which would/could cause antibodies to be made for HMGR?

And thanks, that was a really good find!

Unknown said...

Betsy, I don't know whether the statin fungal agents "carry any of the endotoxins along with them" but what seems to be well validated is that statins increase the endotoxin load by lowering blood cholesterol levels. This is done by blocking the HMG-CoA reductase enzyme.

A steady-high blood sugar level will increase cholesterol production, via generating more acetyl-CoA (assuming no statins or some other agents, like garcinia cambogia extract, are consumed). So, a steady-high blood sugar level will decrease an autoimmune reaction.